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Sample text
26 CPAM III; the lesion is composed of small cystic structures composed of immature bronchioles. There is no alveolar tissue. The cysts neither communicate with the central airways nor the periphery. H&E, ×100 Fig. 27 CPAM III; higher magnification of this lesion showing the immature bronchioles completely covered by Clara cells. H&E, ×250 cystic malformations: CPAM 0 is not cystic and CPAM IV is emphysematous, so we will discuss these lesions under the appropriate term of alveolar dysgenesis and congenital emphysema, although at present we do not have enough data about pathogenesis and the genetic background.
They sit with their long axis firmly attached to the basal membrane and with their side axis provide attachment for several other cells especially for tall columnar cells such as the ciliated and goblet cells. The basal cells are only marginally able to divide and reproduce themselves. R. Johnson, Lovelace Respiratory Research Institute, Albuquerque, NM). Clara cells are one of the main cell types in bronchioles in humans (in some mammals, Clara cells can be found up to the trachea). They together with pneumocytes were for a long time supposed to be the peripheral stem cells (Fig.
14). Pneumocytes type II are capable of regeneration in as far as they are formed out of the peripheral stem cell pool and further on differentiate into type I cells. Stem cells: Only recently it was shown that those peripheral stem cells do exist in niches at the bronchioloalveolar junction. They can be visualized due to their coexpression of stem cell markers CD34 and Oct3/Oct4 together with Clara cell protein 10 and surfactant apoprotein C (also prepro-proteins can be demonstrated) [15–17]. In mouse models using toxicants directed against Clara cells and pneumocytes, it could be shown that the epithelial lining is repopulated by stem cells undergoing differentiation into either pneumocytes type II or Clara cells, respectively [18, 19].
