By Donald Zimmerman MD, Greg Uramoto MD (auth.), A. Wayne Meikle MD (eds.)
A. Wayne Meikle and a distinct panel of professional clinicians convey to undergo their vast wisdom and adventure in dealing with adults and kids present process hormone alternative remedy for the pituitary, parathyroid, thyroid, pancreas, adrenal glands, and gonads. Emphasizing right sufferer administration, the booklet presents to experts and common practitioners alike time-tested, state-of-the-art instructions at the use, tracking, and dosage of hormone therapeutics within the therapy of problems of endocrine functionality, whereas giving an exhaustive research of every remedy. finished and eminently functional, Hormone alternative remedy captures the latest advances in hormone alternative therapeutics and is sure to serve endocrinologists, gynecologists, pediatricians, urologists, internists, and kin practitioners as state-of-the-art typical reference for handling and tracking their patients.
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37. Perez GO, Ostre JR, Robertson GL. Severe hypernatremia with impaired thirst. Am J Nephrol 1989;9:421–434. 38. Nandi M, Harrington AR. Succesful treatment of hypernatremic thirst deficiency with chlorpropamide. Clin Nephrol 1978;10:90–95. 39. Vokes TJ, Gaskill MB, Robertson GL. Antibodies to vasopressin in patients with diabetes insipidus. Ann Inter Med 1988;108:190–195. 40. Iwasacki Y, Oiso Y, Kondo K, et al. Aggravation of subclinical diabetes insipidus during pregnancy. N Engl J Med 1991;324:522–526.
Therefore, treatment is currently limited to sodium restriction in combination with a few drugs that reduce urine output by vasopressin-independent mechanisms. They are: 1. THIAZIDE DIURETICS are the mainstay of therapy for nephrogenic DI. Their pharmacology has been described earlier in the section on pituitary DI. One should remember that thiazide induced hypokalemia may impair glucose tolerance and urinary concentrating ability, either of which could exaggerate polyuria and polydipsia in patients with nephrogenic DI.
Galla JH, Booker BB, Luke RG. Role of loop segment in the concentrating defect of hypercalcemia. Kidney Intl 1986;29(5):977–982. 26. Hebert SC, Brown EM, Harris HW. Role of the Ca(2+)- sensing receptor in divalent mineral ion homeostasis. J Exp Biol 1997;200:295–302. 27. Berl T. The cAMP system in vasopressin-sensitive nephron segments of the vitamin D-treated rat. Kidney Intl 1987;31(5):1065–1071. 28. Beck N, Singh H Reed SW, et al. Pathogenic role of cyclic AMP in the impairment of urinary concentrating ability in acute hypercalcemia.
