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Gastrointestinal Oncology-Evidence and Analysis by Peter McCulloch

By Peter McCulloch

GASTROINTESTINAL surgical procedure: PATHOPHYSIOLOGY AND administration is a useful reference textual content for surgeons and surgical trainees. Written fullyyt by means of Dr. Haile T. Debas, Dean of college of drugs and previous Professor and Chairman, division of surgical procedure, on the collage of California, San Francisco, this ebook offers the unified process merely present in a single-authored textual content. in response to the 3 pillars of knowing specific disorder processes-- primary anatomy and body structure, pathopysiology, and scientific correlations--surgical remedy of GI sickness is gifted because the capacity to right irregular body structure and fix overall healthiness. utilizing an built-in process of easy technology and medical perform, Dr. Debas permits the coed of surgical procedure to achieve an reliable figuring out of ailment methods that minimizes the necessity for rote memorization.

Thirteen lavishly illustrated chapters hide the GI process in addition to gastrointestinal peptides and peptide-secreting tumors, belly trauma, the stomach wall, surgical procedure for morbid weight problems, the liver, spleen, retroperitoneum, and perioperative care. Dozens of precis tables all through each one bankruptcy condense "essentials" for fast reference. complete discussions of medical issues assessment the surgical procedure ideas. With four hundred illustrations and tables, together with countless numbers of anatomical line drawings commissioned solely for this textbook, GASTROINTESTINAL surgical procedure presents a scientific and healing method of surgical illnesses with the intention to turn out priceless to the surgical trainee and the practitioner getting ready for recertification.

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Scand J Gastroenterol 2001; 36:897–903. 50. Thun MJ, Namboodiri MM, Calle EE, Flanders WD, Heath CW, Jr. Aspirin use and risk of fatal cancer. Cancer Res 1993; 53:1322–1327. 51. Funkhouser EM, Sharp GB. Aspirin and reduced risk of oesophageal adenocarcinoma. Cancer 1995; 76:1116–1119. 52. Farrow DC, Vaughan TL, Hansten PD, Stanford JL, Risch HA, Gammon MD. Use of aspirin and other nonsteroidal anti-inflammatory drugs and risk of esophageal and gastric cancer. Cancer Epidemiol Biomarkers Prev 1998; 2:97–102.

Cancer Res 2000; 60:5767–5772. 71. Sharma VK, Crowell MD, Howden CW. Infection with H. pylori or CAGA+ H. pylori is protective against Barrett’s oesophagus (BE): and esophageal adenocarcinoma (EAC): A meta-analysis. Gastroenterology 2004; 126(suppl 2): A-87 [Abstract 452]. 72. Lanas AI, Ortega J, Sopena F. Long-term effects of COX-2 inhibitor on oesophageal epithelial proliferation of patients with Barrett’s esophagus: Preliminary results of a randomized controlled trial. Gastroenterol 2004; 126(suppl 2):A-25.

Most research into the role of COX-2 inhibition in carcinogenesis has concentrated on animal studies and human colonic cancers. The hypothesis for COX-2 inhibition as a preventative strategy for esophageal adenocarcinoma originated from supportive evidence for its role in colonic cancer (31,32). The implication is that the studies and mechanisms discussed in this chapter may be transferable in our understanding of esophageal carcinoma. Indeed, COX-2 inhibition has been shown to reduce the development of esophageal adenocarcinoma in animal models (33).

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