By Philip Rubin, L.S. Constine, Lawrence B. Marks, Paul Okunieff
This quantity is predicated at the LENT V NCI-sponsored assembly held in may possibly 2004 and the CURED I convention in 2006. Written via specialists within the box, it addresses a serious issues when it comes to past due results, akin to mechanisms of damage, the function of screening, thoughts for interventions, moment malignancies, and prevention. it's was hoping that those findings can help readers to avoid and deal with the long term side-effects of irradiation.
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Additional resources for CURED I - LENT Late Effects of Cancer Treatment on Normal Tissues (Medical Radiology Radiation Oncology)
Endothelial cells (EC) are key elements of the vessel wall, present at all levels of the vascular tree, and their integrity is essential for vascular function . This description summarizes in vivo and in vitro data indicating the role of EC in the pathologic processes produced by ionizing radiation. 19 3 20 L. F. 2 Physiology of Endothelial Cells A discussion of the radiation effects on endothelial cells should be preceded by some review of the complex physiology of the endothelium . g. g.
Groups of 10 ICAM-1 –/– and ICAM1 +/+ mice were irradiated at the doses indicated. Those animals that survived for 18 months post-irradiation were sacriﬁced, and lungs were prepared for sectioning. Alveolar septal wall thickness was measured for ﬁve sections in each group. Fold increase in wall thickness normalized to non-irradiated mice is shown. Asterisk indicates statistical signiﬁcance. (Reprinted from  with permission of Oxford University Press) context of radiation resistance. However, PI3K inhibition can also be applied to the prevention and treatment of radiation-induced pneumonitis, as described below.
The production of ICAM-1 increases substantially immediately following irradiation of mouse lung (four-fold within 2 days of treatment). However, it is not until day 28 post-irradiation that ICAM-1 production peaks (Fig. 1). This late increase in ICAM-1 is reﬂective of the delayed nature of radiation ﬁbrosis and pneumonitis. It is felt that the ICAM-I production provides a place of attachment for leukocytes that are recruited to the vicinity that essentially provides a positive feedback loop. It should be noted that studies using a pig model have suggested less of a role for ICAM-1.