By Icon Health Publications
This can be a 3-in-1 reference publication. It provides a whole scientific dictionary masking hundreds of thousands of phrases and expressions in terms of arrhythmias. It additionally offers broad lists of bibliographic citations. ultimately, it offers details to clients on the best way to replace their wisdom utilizing numerous web assets. The e-book is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to get to grips with study devoted to arrhythmias. in the event that your time is effective, this e-book is for you. First, you won't waste time looking the net whereas lacking loads of appropriate details. moment, the booklet additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing enormous quantities of websites.
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Extra resources for Arrhythmias - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
Example text
During VT, the ventricles lack sufficient time to fill with blood prior to each contraction often resulting in dizziness, loss of consciousness and sudden cardiac arrest. Catheter ablation has been shown to be an effective means for curing many arrhythmias, but current approaches are not able to coagulate tissue in the midmyocardium or subepicardial regions where foci responsible for VT often originate. We have developed a cooled-tip laser catheter (CTLC) capable of creating large lesions that extend into these regions with little to no thermal damage to the endocardium.
We will test the hypothesis that, in the setting of an ischemically induced spontaneous arrhythmia, a defibrillation shock must do three things: (1) stop all fibrillation wavefronts, (2) not restart fibrillation, and (3) stop the trigger of the original arrhythmia. We will test the hypothesis that a major determinant of whether or not a spontaneous arrhythmia, a defibrillation shock must do three things: (1) stop all fibrillation wavefronts, (2)m not restart fibrillation and (3) stop the trigger of the original.
Specifically, we will ask: What determines the limited expression patterns of two specialized cardiovascular connexins (Cx37 and Cx40)? What is the extent of natural variation of cardiovascular connexin sequences; what is the prevalence of different polymorphic variants; are they associated with clinical disease; and, do they exhibit altered channel function? What sequence features determine the molecular permeability of connexin 34 Arrhythmias channels? We will use a variety of molecular tools to analyze the promoters for Cx37 and Cx40 in vitro and in vivo.
